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ca  (Worthington Biochemical)


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  • 99

    Structured Review

    Worthington Biochemical ca
    Ca, supplied by Worthington Biochemical, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/ca/ca/custom%40ca%4042607499
    Average 99 stars, based on 1 article reviews
    ca - by Bioz Stars, 2026-09
    99/100 stars

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    Related Articles

    Adsorption:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Virus:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Staining:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Chromatin Immunoprecipitation:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Immunoprecipitation:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Extraction:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Whole Genome Amplification:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Protein Extraction:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    In Vitro:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Mouse Assay:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Single Cell:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Isolation:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Suspension:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Activity Assay:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Modification:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g

    Imaging:

    Article Title: Myocilin drives cardiomyocyte mitochondrial dysfunction via SLC3A2-dependent redox imbalance in heart failure
    Article Snippet: rtic cannulation and retrograde perfusion at 37°C. The hearts were initially perfused with Ca 2+ -free Tyrode's solution for 3 min, followed by enzymatic digestion using type II collagenase (300 U/mL; Worthington Biochemical Corporation, Lakewood, NJ, USA). Calcium was gradually reintroduced during digestion until the hearts became pale and flaccid. The ventricles were then dissected, minced, and g



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